Wikipedia

ISHAR as Wikipedia Representative

ISHAR’s researchers include Wikipedians who are responsible for improving the encyclopedia in strict accordance with the highest ideals of the spirit and letter of Wikipedia’s guidelines, acting under the principles of Wikipedia GLAM.  ISHAR is not in opposition or competition with Wikipedia, but is designed to be complementary.   ISHAR’s 25,000+ sources are all designed to be easily formatted into Wikipedia references.

Specifically these guidelines require us to list source type with an archived reference, reveal any ‘conflict of interest’ on any page or article where appropriate, and present sources to Wikipedia editors working on the article.

ISHAR does not promote, advocate, or market material on Wikipedia.  ISHAR’s goal on Wikipedia is to provide reliable academic sourcing to help promote accurate, academic representation of Integrative Studies.

If there are questions and concerns about ISHAR Wikipedians, or for any other questions, please contact [email protected]

 Please note that Wikipedia user SAS81 is no longer affiliated with ISHAR and does not represent ISHAR on Wikipedia.
 

Below is a sample of an ISHAR library source, and below it a Wikipedia-formatted reference of the same source:

 

  • Caloric intake and aging: mechanisms in rodents and a study in nonhuman primates

    Type Journal Article
    Author J. Wanagat
    Author D. B. Allison
    Author R. Weindruch
    Volume 52
    Issue 2 Suppl
    Pages 35-40
    Publication Toxicological Sciences: An Official Journal of the Society of Toxicology
    ISSN 1096-6080
    Date Dec 1999
    Extra PMID: 10630588
    Journal Abbr Toxicol. Sci.
    Library Catalog NCBI PubMed
    Language eng
    Abstract Caloric restriction (CR) increases maximum life span in rodents while attenuating the development of age-associated pathological and biological changes. Although nearly all of the rodent studies have initiated CR early in life (1-3 months of age), CR, when started at 12 months of age, also extends maximum life span in mice. Two main questions face investigators of CR. One concerns the mechanisms by which CR retards aging and diseases in rodents. There is evidence that CR may act, at least in part, by reducing oxidative stress. A CR-induced decrease in oxidative stress appears to be most profound in post-mitotic tissues and may derive from lower mitochondrial production of free radicals. The second issue is whether CR will exert similar effects in primates. Studies on CR in rhesus monkeys (maximum life span approximately 40 years) support the notion of human translatability. We describe the University of Wisconsin Study of rhesus monkeys subjected to a 30% reduction of caloric intake starting at either 1989 or 1994 when they were approximately 10 years old. The data from our study and from other trials suggest that CR can be safely carried out in monkeys and that certain physiological effects of CR that occur in rodents (e.g., decreased blood glucose and insulin levels, improved insulin sensitivity, and lowering of body temperature) also occur in monkeys. Whether oxidative stress in monkeys is reduced by CR will be known by the year 2000, while effects on longevity and diseases should be clearly seen by, appropriately, 2020.
    Short Title Caloric intake and aging
    Date Added 10/2/2014, 9:19:18 PM
    Modified 10/2/2014, 9:19:18 PM

    Tags:

    • Aging
    • Animals
    • Energy Intake
    • Humans
    • Longevity
    • Macaca mulatta
    • Mice

Wikipedia Formatting:

{{Cite journal
| issn = 1096-6080
| volume = 52
| issue = 2 Suppl
| pages = 35–40
| last = Wanagat
| first = J.
| coauthors = D. B. Allison, R. Weindruch
| title = Caloric intake and aging: mechanisms in rodents and a study in nonhuman primates
| journal = Toxicological Sciences: An Official Journal of the Society of Toxicology
| date = 1999-12
| pmid = 10630588
}}

Formatted Wikipedia Reference:

  • Wanagat, J.; D. B. Allison, R. Weindruch (1999-12). “Caloric intake and aging: mechanisms in rodents and a study in nonhuman primates”. Toxicological Sciences: An Official Journal of the Society of Toxicology 52 (2 Suppl): 35–40. ISSN 1096-6080.PMID 10630588.

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